As caretakers of horses we have a responsibility to
give them a safe and healthy life. This is sometimes hard to do, given
that we may not own them for their whole lives or even half. With
slaughterhouses closed and the market being what it is today, there are
more horses in our care than usual. This leads to some serious thinking
about how everyone can do their part to ensure a good quality of life.
Valued animals typically are taken better care of throughout life even
if they change hands a few times. I’m talking not only about monetary
value but also value in the eye of the beholder. This is made up of a
combination of the human animal bond and the degree to which a horse
performs the tasks expected of him. Two ways we can create a valuable
horse are with good breeding and good training.
Breeding is a big responsibility. Physical ability
and beauty are important but so is avoiding a serious genetic flaw.
Adding all the good qualities of two horses and neglecting to account
for the bad will not give favorable results. Admittedly, finding out
the bad qualities may be harder to do because they are not advertised.
Many common problems in horses have some genetic
basis. This is easily demonstrated by the fact that certain breeds are
associated with certain problems suggesting that it is a familial
trait. This doesn’t mean to stop breeding purebloods. It means start
trying to figure out the family lines that have a low incidence of the
problem. No “good deal” on a stud fee will ever make up for the cost of
treating a disease that was inherited from that stallion.
Some diseases take years to manifest so you may
need to look beyond the futurity years to get the full picture. Other
diseases are fatal at a young age but can still occur when two parents
with the same hidden trait are combined. Certain diseases have been
linked to a specific gene and can be tested for but for those that
don’t, it may require a family history to check for repeat incidence.
Often diseases are highly, but not exclusively, associated with a
physical trait that can be observed with knowledge of conformation or
body type.
The following table is a list of several diseases
that can be inherited.
|
Disease name and description. |
Associated physical trait/s. |
Breed commonly affected. |
Genetic testing available |
|
Brachygnathia
aka Parrot mouth
-Top jaw longer than the bottom jaw causing
eating problems since teeth don’t align. |
Top front teeth override bottom front
teeth. |
None more than others. |
No |
|
Cerebellar Abiotrophy
-Lack of control of movements due to under
developed portion of the brain. |
Tremors and exaggerated, jerky movements. |
Arabian Horses |
No |
|
Chronic Obstructive Pulmonary Disease
(COPD) aka Heaves
-Allergy associated inflammation and
scarring of the lungs causing respiratory problems.
|
Abdominal muscle near flank becomes
prominent after a while due to the effort of breathing.
Commonly called a heave line. |
Studies have shown it runs in families but
not a specific breed. |
No |
|
Cryptorchid
-Failure of one testicle to descend into
the scrotem. |
Only one testicle in the scrotem. |
Can be in any breed but prominent in
Miniature Horses |
No |
|
Developmental Orthopedic Diseases (DOD)
-Includes many conditions: osteochondrosis,
osteochondritis desicans, angular limb deformities, flexural
deformities, subchondral cysts, stenotic myelopathies, cuboidal
bone malformations, and physitis |
Depends on the specific disease process. |
Predominantly in purebreds. Most commonly
affected are Thoroughbreds, Quarter Horses, Standardbreds, and
Arabians. |
No |
|
Glycogen Branching Enzyme Deficiency
(GBED)
-Muscle disease that is fatal with in 18
weeks of life. |
Foals are weak and may have flexural
deformities. Parents are carriers but appear normal. |
Quarter Horses and Paints |
Yes
|
|
Equine hyperelastosis cutis (HC)
Aka HERDA
-Skin separates between the deep and
superficial layers resulting in "mushy" or "stretchy" skin that
is not securely attached in multiple areas. |
Skin can be abnormally stretched and sores
may develop in areas that are rubbed from tack or blankets. |
Poco Bueno/King bloodline of Quarter Horses
and horses with Quarter Horse lineage, such as Paints and
Appaloosas. |
No |
|
Hyperkalemic Periodic Paralysis (HYPP)
-Muscle disorder causing intermittent
muscle tremors and weakness.
|
Often have heavy muscling. |
Seen only in descendents of the Quarter
Horse sire Impressive. |
Yes |
|
Laminitis
Aka founder
-Inflammation and pain of the tissue
|
There are several causes for laminitis but
cresty necked, over weight, easy keepers are more prone and tend
to be more chronic with repeated episodes. |
Can be in any breed but prominent in
Morgans and ponies. |
No |
|
Lavender Foal Syndrome
-Fatal neurologic disease causing seizures
and inability to stand. |
Dilute gray color hair coat that appears
lavender. |
Egyptian Arabians |
No |
|
Lethal White Syndrome
-Fatal defect of the gastrointestinal tract
that causes colic within 24-48 hours of life. |
All white foal born to frame overo color
patterned parents. |
Paints |
Yes |
|
Narcolepsy
-Uncontrolled episodes of loss of muscle
tone and sleep. |
None |
Suffolk and Shetland pony foals |
No |
|
Navicular disease (syndrome)
-Small pivotal bone in the foot develops
degenerative changes and lameness. |
Horses with smaller than average feet for
their body size. |
Quarter Horses, Appaloosas, Paints and some
warmbloods more often have the predisposing conformation. |
No |
|
Occipito-atlanto-axial Malformation
-Causes instability of neck joints due to
malformed skull and vertebrae. |
It is a cause of wobblers. |
Arabian Horses |
No |
|
Patellar luxation
-Small bone that normally sits in groove
over stifle slips out of place. |
Crouched appearance of hind limbs. |
Miniature Norses |
No |
|
Polysaccaride Storage Myopathy (PSSM)
-Muscle disorder that causes muscle
destruction and pain that sometimes mimics colic. Happens at
the start of training or after a period of rest.
|
None |
Described in Quarter horses, warmbloods and
draught breeds. Evidence of family line association in the QH. |
Genetic testing in the process of being
devel-oped. |
|
Recurrent Exertional Rhabdomyolysis
(RER)
- Muscle disorder that causes muscle
stiffness preceded by excitement and stress. |
None |
Occurs commonly in young nervous
Thoroughbred, Standardbred and Arabian Horses.
|
No |
|
Severe Combined Immune Deficiency
Syndrome (SCIDS)
-Disorder of the immune system leading to
severe and fatal infections by 5 months of age. |
None |
Arabian Horses |
Yes |
|
Equine Recurrent Uveitis (ERU)
Aka Moon Blindness
-Episodes of inflammation in the eye that
can cause degeneration of the eye and eventual blindness. |
During episodes eye(s) may have a cloudy
blue appearance. |
Can occur in any breed but Appaloosas are
8.3 times as likely to develop it and more often blinded by it. |
No |
In those diseases for which genetic testing is
available, the affected animal can be tested to confirm a diagnosis, but
even more importantly prospective dams and sires may be tested prior to
breeding to check for carrier status. This can help avoid breeding two
horses whose combined genetics would result in a disease.
If you’re planning to breed, be sure to do your
homework. Inability to continue as a performance horse due to heaves or
laminitis is not a good reason to be a broodmare in retirement. This
can only propagate the problem and do harm to the future offspring and
the breeders reputation.
The second part of value is training. Gone are the
days when just being a horse made them valuable. They are a dime a
dozen right now unless they have a high level of training or superior
bloodlines Many people are looking for the horse they can get on after
work or on the weekend and have a good ride. With so many options,
bloodlines alone may not be a strong enough selling point. Prospective
buyers can easily find an equally bred horse that doesn’t cost them
additional time and money on sending it somewhere for training.
What I’m advocating for is to invest in your
horses’ future with age appropriate training. A yearling should be
halter broke and willing to allow their feet and legs to be dealt with
(just ask any veterinarian of farrier). A two year old may or may not
start under saddle but still needs to be handled to improve manners and
learn to stand quietly. By three years old most can be started under
saddle and if you don’t have experience or a training mentor, a
professional trainer is worth the investment. Well-mannered, amiable
horses are less likely to end up in the unwanted population later in
life. Our lives’ move fast and sometimes things prevent us from working
with our horses, but it’s a worthy goal.
In a market flooded with horses, many are suffering
from neglect. Careful and well thought out selection of breeding stock
along with a little training time could greatly help reduce the number
of unwanted horses in the future.